Search results for "genetics [Transcriptome]"

showing 10 items of 3033 documents

3D molecular phenotyping of cleared human brain tissues with light-sheet fluorescence microscopy

2022

AbstractThe combination of optical tissue transparency with immunofluorescence allows the molecular characterization of biological tissues in 3D. However, adult human organs are particularly challenging to become transparent because of the autofluorescence contributions of aged tissues. To meet this challenge, we optimized SHORT (SWITCH—H2O2—antigen Retrieval—TDE), a procedure based on standard histological treatments in combination with a refined clearing procedure to clear and label portions of the human brain. 3D histological characterization with multiple molecules is performed on cleared samples with a combination of multi-colors and multi-rounds labeling. By performing fast 3D imaging…

AdultImaging Three-DimensionalMicroscopy FluorescenceBrainFluorescent Antibody TechniqueHumansMedicine (miscellaneous)Hydrogen PeroxideGeneral Agricultural and Biological SciencesSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)General Biochemistry Genetics and Molecular BiologyAgedCommunications Biology
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LPS-mediated production of pro/anti-inflammatory cytokines and eicosanoids in whole blood samples: Biological effects of +896A/G TLR4 polymorphism in…

2011

Toll-like receptors (TLRs) are the principal mediators of rapid microbial recognition: the lipopolysaccharide (LPS) receptor TLR4 seems to have a paradigmatic role. Single nucleotide polymorphisms (SNPs) in the TLR4 gene, such as +896A/G, known to attenuate receptor signaling, have been described. The +896A/G SNP is significantly less frequent in patients with myocardial infarction, Alzheimer's disease or prostate cancer, whereas it is overrepresented in centenarians. To clarify and confirm the biological effects of +896A/G SNP and its role in the pathophysiology of age-related diseases and longevity, we assessed the levels of IL-6, TNF-α, IL-10 and eicosanoids (LTB4 and PGE2) in LPS-stimul…

AdultLipopolysaccharidesMaleAgingAgeing Cytokines Eicosanoids Genetics Inflammation Longevity TLR4PopulationInflammationSingle-nucleotide polymorphismBiologyLeukotriene B4Polymorphism Single NucleotideDinoprostonemedicineHumansSNPeducationReceptorSettore MED/04 - Patologia Generaleeducation.field_of_studyMiddle AgedToll-Like Receptor 4ItalyEicosanoidImmunologyTLR4CytokinesFemalelipids (amino acids peptides and proteins)medicine.symptomDevelopmental BiologyEicosanoid ProductionMechanisms of Ageing and Development
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Role of TLR4 polymorphisms in inflammatory responses: implications for unsuccessful aging.

2007

The total burden of infection at various sites may affect the progression of atherosclerosis and Alzheimer's disease (AD), the risk being modulated by host genotype. The role of lipopolysaccharide (LPS) receptor TLR4 is paradigmatic. It initiates the innate immune response against gram-negative bacteria, and TLR4 single nucleotide polymorphisms (SNPs), such as +896A/G, known to attenuate receptor signaling, have been described. This SNP shows a significantly lower frequency in patients affected by myocardial infarction or AD. Thus, people genetically predisposed to developing lower inflammatory activity seem to have less chance of developing cardiovascular disease (CVD) or AD. In the presen…

AdultLipopolysaccharidesMaleAgingTime FactorsLipopolysaccharideGenotypeLeukotriene B4Myocardial InfarctionInflammationSingle-nucleotide polymorphismBiologyLeukotriene B4Polymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyDinoprostoneProinflammatory cytokinechemistry.chemical_compoundHistory and Philosophy of ScienceAlzheimer DiseaseGenotypemedicineTLR4 SNPAgeing related disease longevityEscherichia coliHumansCells CulturedEscherichia coli InfectionsSettore MED/04 - Patologia GeneraleInflammationInnate immune systemBlood CellsGeneral NeuroscienceMiddle AgedImmunity InnateToll-Like Receptor 4chemistryImmunologyTLR4lipids (amino acids peptides and proteins)Femalemedicine.symptomAnnals of the New York Academy of Sciences
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Inflammation, Longevity, and Cardiovascular Diseases: Role of Polymorphisms of TLR4

2006

The total burden of infection at various sites may affect the progression of atherosclerosis, the risk being modulated by host genotype. The role of lipopolysaccaride receptor TLR4 is paradigmatic. It initiates the innate immune response against gram-negative bacteria; and TLR4 polymorphisms, as ASP299GLY, suggested to attenuate receptor signaling, have been described. We demonstrated that TLR4 ASP299GLY polymorphism shows a significantly lower frequency in patients affected by myocardial infarction compared to controls, whereas centenarians show a higher frequency. Thus, people genetically predisposed to developing weak inflammatory activity, seem to have fewer chances of developing cardio…

AdultLipopolysaccharidesMaleHeterozygoteTime Factorsmedia_common.quotation_subjectmedicine.medical_treatmentLongevityMyocardial InfarctionEnzyme-Linked Immunosorbent AssayInflammationBiologyGeneral Biochemistry Genetics and Molecular BiologyAMIHistory and Philosophy of SciencemedicineHumansGenetic Predisposition to DiseaseTLR4Interleukin 6media_commonInflammationPolymorphism GeneticInnate immune systemInterleukin-6General NeuroscienceLongevityInterleukinHeterozygote advantageMiddle AgedToll-Like Receptor 4CytokineAcute DiseaseMutationImmunologyTLR4biology.proteinFemalemedicine.symptomAnnals of the New York Academy of Sciences
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Blood coagulation factor XII drives adaptive immunity during neuroinflammation via CD87-mediated modulation of dendritic cells

2016

Aberrant immune responses represent the underlying cause of central nervous system (CNS) autoimmunity, including multiple sclerosis (MS). Recent evidence implicated the crosstalk between coagulation and immunity in CNS autoimmunity. Here we identify coagulation factor XII (FXII), the initiator of the intrinsic coagulation cascade and the kallikrein–kinin system, as a specific immune cell modulator. High levels of FXII activity are present in the plasma of MS patients during relapse. Deficiency or pharmacologic blockade of FXII renders mice less susceptible to experimental autoimmune encephalomyelitis (a model of MS) and is accompanied by reduced numbers of interleukin-17A-producing T cells.…

AdultMale0301 basic medicineEncephalomyelitis Autoimmune ExperimentalMultiple Sclerosisanimal structuresT-LymphocytesScienceMedizinGeneral Physics and AstronomyKininsCoagulation Factor XIIAdaptive ImmunityBiologymedicine.disease_causeArticleGeneral Biochemistry Genetics and Molecular BiologyReceptors Urokinase Plasminogen ActivatorAutoimmunityYoung Adult03 medical and health sciencesImmune systemddc:570medicineAnimalsHumansddc:610cardiovascular diseasesNeuroinflammationAgedFactor XIIMultidisciplinaryInterleukin-17QExperimental autoimmune encephalomyelitisCell DifferentiationDendritic CellsGeneral ChemistryMiddle Agedmedicine.diseaseAcquired immune systemMice Inbred C57BL030104 developmental biologyNeuroimmunologyFactor XIIImmunologyFemaleKallikreinscirculatory and respiratory physiologyNature Communications
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Second line therapy with axitinib after only prior sunitinib in metastatic renal cell cancer: Italian multicenter real world SAX study final results

2019

Abstract Background This multi-institutional retrospective real life study was conducted in 22 Italian Oncology Centers and evaluated the role of Axitinib in second line treatment in not selected mRCC patients. Methods 148 mRCC patients were evaluated. According to Heng score 15.5%, 60.1% and 24.4% of patients were at poor risk, intermediate and favorable risk, respectively. Results PFS, OS, DCR and ORR were 7.14 months, 15.5 months, 70.6% and 16.6%, respectively. The duration of prior sunitinib treatment correlated with a longer significant mPFS, 8.8 vs 6.3 months, respectively. Axitinib therapy was safe, without grade 4 adverse events. The most frequent toxicities of all grades were: fati…

AdultMale0301 basic medicineOncologymedicine.medical_specialtyMultivariate analysisAxitinibAxitinib; Metastatic; Renal cancer; Sunitinib; Treatmentmedicine.medical_treatmentPopulationlcsh:MedicineKaplan-Meier EstimateDisease-Free SurvivalGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineInternal medicineSunitinibHumansMedicineNeoplasm MetastasiseducationAdverse effectMetastatic renal cell cancerCarcinoma Renal CellAgedAged 80 and overSecond-line therapyeducation.field_of_studybusiness.industrySunitinibResearchlcsh:RGeneral MedicineMiddle AgedKidney NeoplasmsNephrectomyAxitinibTreatment030104 developmental biologyRenal cancer030220 oncology & carcinogenesisMultivariate AnalysisMetastaticFemalebusinessmedicine.drugJournal of Translational Medicine
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Elevated levels of Bcl-3 inhibits Treg development and function resulting in spontaneous colitis

2017

Bcl-3 is an atypical NF-κB family member that regulates NF-κB-dependent gene expression in effector T cells, but a cell-intrinsic function in regulatory T (Treg) cells and colitis is not clear. Here we show that Bcl-3 expression levels in colonic T cells correlate with disease manifestation in patients with inflammatory bowel disease. Mice with T-cell-specific overexpression of Bcl-3 develop severe colitis that can be attributed to defective Treg cell development and function, leading to the infiltration of immune cells such as pro-inflammatory γδT cells, but not αβ T cells. In Treg cells, Bcl-3 associates directly with NF-κB p50 to inhibit DNA binding of p50/p50 and p50/p65 NF-κB dimers, t…

AdultMale0301 basic medicineP50ScienceGeneral Physics and AstronomyBiologyT-Lymphocytes RegulatoryInflammatory bowel diseaseArticleGeneral Biochemistry Genetics and Molecular BiologyYoung Adult03 medical and health sciences0302 clinical medicineImmune systemB-Cell Lymphoma 3 ProteinProto-Oncogene ProteinsGene expressionmedicineAnimalsHumansColitisMice KnockoutRegulation of gene expressionMultidisciplinaryEffectorHEK 293 cellsQNF-kappa BTranscription Factor RelANF-kappa B p50 SubunitGeneral ChemistryMiddle AgedColitismedicine.diseaseMice Inbred C57BLHEK293 Cells030104 developmental biologyGene Expression Regulation030220 oncology & carcinogenesisImmunologyFemaleProtein BindingTranscription FactorsNature Communications
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Metabolic adaptation in the human gut microbiota during pregnancy and the first year of life

2018

Abstract Background The relationship between the gut microbiome and the human host is dynamic and we may expect adjustments in microbiome function if host physiology changes. Metatranscriptomic approaches should be key in unraveling how such adjustments occur. Methods We employ metatranscriptomic sequencing analyses to study gene expression in the gut microbiota of infants through their first year of life, and of their mothers days before delivery and one year afterwards. Findings In infants, hallmarks of aerobic metabolism disappear from the microbial metatranscriptome as development proceeds, while the expression of functions related to carbohydrate transport and metabolism increases and …

AdultMale0301 basic medicineResearch paperCarbohydrate transportPregnancy Trimester ThirdPhysiologyFirst year of lifeButyrateGut floraGeneral Biochemistry Genetics and Molecular BiologyFeces03 medical and health sciences0302 clinical medicineBacterial ProteinsPregnancymedicineHumansGutMicrobiomeMetatranscriptomicsPregnancyBacteriabiologySequence Analysis RNAGene Expression ProfilingMicrobiotaInfant NewbornInfantGene Expression Regulation BacterialGeneral MedicineMetabolismLipid Metabolismbiology.organism_classificationmedicine.diseaseIntestinal epitheliumGastrointestinal MicrobiomeButyratesMetabolism030104 developmental biology030220 oncology & carcinogenesisFemaleMaternal AgeEBioMedicine
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Erratum to: Donor age and long-term culture do not negatively influence the stem potential of limbal fibroblast-like stem cells

2016

In regenerative medicine the maintenance of stem cell properties is of crucial importance. Ageing is considered a cause of reduced stemness capability. The limbus is a stem niche of easy access and harbors two stem cell populations: epithelial stem cells and fibroblast-like stem cells. Our aim was to investigate whether donor age and/or long-term culture have any influence on stem cell marker expression and the profiles in the fibroblast-like stem cell population.Fibroblast-like stem cells were isolated and digested from 25 limbus samples of normal human corneo-scleral rings and long-term cultures were obtained. SSEA4 expression and sphere-forming capability were evaluated; cytofluorimetric…

AdultMale0301 basic medicineStage-Specific Embryonic AntigensPrimary Cell CultureGene ExpressionMedicine (miscellaneous)Limbus CorneaeBiologyBiochemistry Genetics and Molecular Biology (miscellaneous)Donor age03 medical and health sciencesCell MovementSpheroids CellularmedicineATP Binding Cassette Transporter Subfamily G Member 2HumansFibroblastAgedCell ProliferationStem CellsAge FactorsEpithelium CornealCell DifferentiationEpithelial CellsHLA-DR AntigensNanog Homeobox ProteinCell BiologyFibroblastsMiddle AgedMolecular medicinehumanitiesNeoplasm ProteinsCell biology030104 developmental biologymedicine.anatomical_structureLeukocyte Common AntigensMolecular MedicineFemaleErratumStem cellOctamer Transcription Factor-3BiomarkersStem Cell Research & Therapy
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Low Prevalence of Lactase Persistence in Bronze Age Europe Indicates Ongoing Strong Selection over the Last 3,000 Years

2020

Lactase persistence (LP), the continued expression of lactase into adulthood, is the most strongly selected single gene trait over the last 10,000 years inmultiple human populations. It has been posited that the primary allele causing LP among Eurasians, rs4988235-A [1], only rose to appreciable frequencies during the Bronze and Iron Ages [2, 3], long after humans started consuming milk from domesticated animals. This rapid rise has been attributed to an influx of people from the Pontic-Caspian steppe that began around 5,000 years ago [4, 5]. We investigate the spatiotemporal spread of LP through an analysis of 14 warriors from the Tollense Bronze Age battlefield in northern Germany ( 3,20…

AdultMale0301 basic medicineSteppemedicine.medical_treatmentBiologyengineering.materialDNA MitochondrialWhite PeopleGeneral Biochemistry Genetics and Molecular BiologyYoung Adult03 medical and health sciences0302 clinical medicineGene FrequencyBronze AgeLactase persistenceHuman population geneticsmedicineHumansDNA AncientSelection GeneticBronzeDomesticationancient DNALactasegeographygeography.geographical_feature_categoryLactaseBody RemainsEuropeLactase persistence030104 developmental biologyIron AgeengineeringFemaleGeneral Agricultural and Biological Sciences030217 neurology & neurosurgeryDemography
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